Abstract
In this study we have examined the results of salmon calcitonin treatment on migraine pain. The mechanism by which calcitonin induces analgesia is still not understood. We observed the effect of a 5-day treatment with salmon calcitonin (IM 100 IU day ) on circulating levels of β-endorphin, ACTH, and cortisol in 20 patients with migraine during the headache-free period. All 3 hormones were increased after the calcitonin administration and the maximum increase was obtained in β-endorphin levels. There were significant statistical correlations between β-endorphin, ACTH, and cortisol levels determined before and after calcitonin treatment. calcitonin/migraine/β-endorphin
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