Abstract
Patients receiving large amounts of penicillin intravenously develop a fast albumin component, which disappears after therapy is discontinued. In vitro experi- ments using normal sera, sera from patients with known inherited albumin variants, and purified albumin solutions show that this phenomenon is related to the concen- tration of the drug in the serum and that the abnormal component is the result of binding of the drug to albumin.
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