Abstract
The transduction of the gal + character into gal − mutants of Escherichia coli K12 with high frequency transducing (HFT) lysates of phage λ has been studied. Sensitive recipients infected at low multiplicity give rise to heterogenotes carrying a defective prophage. Simultaneous infection with a non-transducing phage increases the probability of transduction and causes the cell to become lysogenic. Lysogenic recipients upon transduction generally remain lysogenic for the original prophage. Prophage substitution and double lysogenization rarely occur. Loss of the gal + character from the defective lysogenics is generally accompanied by loss of the defective prophage. Loss of the gal + character from the double lysogenics is correlated with changes in prophage content.
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