Abstract
Background and Aims: The current definition of an advanced atherosclerotic plaque type is predominantly driven by pathology-based descriptions. Histo-pathological studies have been instrumental in revealing key processes leading to plaque thrombosis. However, the diversity of pathology-based lesion types demands the improvement of the discriminative value to identify the arterial segment at risk for a major thrombotic event. We hypothesized that transcriptomic analyses of plaques provide major added value to identify lesions at risk for a major thrombotic event and underlying pathogenetic mechanisms.
Published Version
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