Abstract
Purpose: Recent developments have demonstrated that intrinsic cartilage repair is possible in certain osteoarthritis (OA) patients. A growing body of literature suggests an influence of joint microenvironment on intrinsic cartilage repair via affecting chondrogenic capacity of stem cells; i.e. this environment can be permissive or non-permissive for repair. A method able to discriminate between patients with permissive or non-permissive articular joint microenvironments would be of great use to predict the outcome of cartilage repair therapies.
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