Abstract
Hypoxic-ischemic injury can produce diverse patterns of brain damage detected on MR imaging which mainly depend on the severity of brain ischemia and the maturity of the brain at the time of insult (1). In a term infant severe ischemia produces maximum damage in the lateral thalami, posteromedial putamen, hippocampi and the dorsal brain stem in addition to the perirolandic cortex (2). This is because these areas are the most metabolically active sites in the neonate (3).
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