Abstract

In an earlier article,1 I proposed a theory and described the supporting evidence for the pathways for the possible influence of tumour necrosis factor α (TNFα) on maternal breast milk and its influence on the pathophysiology of infantile colic. I emphasised how the increased TNF levels of a breastfeeding mother affect TNF, melatonin and serotonin levels of a colicky baby, and examined evidence for the direct and indirect regulatory effects of acupuncture over TNFα, serotonin and melatonin levels, and thereby infantile colic. In a commentary on my paper,2 Reinthal et al stated that ‘Even if TNFα does play a …

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