Abstract
The right half of the hindbrain of Rana pipiens embryos was removed at stage 16 in order to determine whether thyroxine would influence regeneration of the deleted half. Thyroxine was found to accelerate the mitotic activity of the ependyma of both the right regenerating and left intact halves. However, this activity did not enhance restitution of the deleted half. Thyroxine-treated embryos experienced peaks of mitotic activity between stages 24 and 25. The decline which followed reached levels lower than those of controls by stage I. Findings in this study suggest that at embryonic stages exogenous thyroxine merely accelerates mitotic activity in neurons ready or nearly ready to divide and that upon termination of division an overall decline in proliferation occurs due to growth and synthetic processes occurring in daughter cells.
Published Version
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