Abstract
Early complications after Bilroth-II gastric resection for peptic ulcer disease are duodenal stump failure, anastomosis, postoperative pancreatitis, anastomosis stitch failure, etc. First of all duodenostasis underlies these complications, and it appears as a result of duodenum atony and accumulation of bile, pancreatic juice and gastrointestinal contents in it. The resultant increase in hydrostatic pressure in the duodenal cavity can lead to residual limb failure. In addition, increased hydrostatic pressure contributes to inflammatory phenomena in the area of the junction, supports intestinal paresis and, preventing further secretion of pancreatic juice, leads to pancreatitis. Lack of enteral nutrition in the postoperative period disturbs metabolism and reduces plastic capabilities of the body. Stagnation of intestinal contents also has a toxic effect.
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