Abstract
Serial measurements of thoracic gas volume and arterial oxygen tension in a group of small premature infants are reported. The study demonstrated that when thoracic gas volume reached levels for normal, full-term infants arterial oxygen tension approached full-term values. The study indicates extensive pulmonary abnormality in clinically non-distressed premature infants, most likely due to persistent atelectasis or partially aerated alveoli. Observations suggest that the infant attempts to correct this abnormality by frequent periodic hyperinflations or sighs.
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