Abstract

Proximal cervical carotid artery occlusion has become the preferred method for the treatment of symptomatic intracavernous aneurysms (primarily because of the risk to cranial nerve function that accompanies direct cavernous sinus surgery for clipping of such aneurysms). Most patients tolerate cervical internal carotid artery occlusion because the circle of Willis (particularly through the anterior communicating and posterior communicating arteries) provides adequate collateral circulation. However, in perhaps 15% of patients, because of collateral circulation inadequacy, carotid artery occlusion cannot be performed without causing injurious cerebral ischemia. The adequacy of the cerebral collateral circulation is tested, before permanent carotid occlusion, by temporary balloon occlusion performed under local anesthesia. When a patient develops ischemic neurological symptoms (or cerebral blood flow inadequacy measured by transcranial Doppler or xenon-computed tomography [CT]) during 20 to 30 minutes of trial occlusion), it is clear that the patient

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