Abstract
As is common knowledge, there is an argument as to whether or not the toxic radical cyanide is an important aetiological agent responsible for the development of some of the conditions grouped under the term toxic amblyopia. There are arguments and counterarguments, and it would appear useful at this stage to state the evidence for and against cyanide as a cause of these conditions. The argument primarily concerns tobacco amblyopia, but has been enlarged to include certain other conditions including Leber's hereditary optic atrophy and tropical ataxic neuropathy. In all of these conditions the functional loss is due to demyelination (Sachs, I887; Rehsteiner, I930). In tobacco amblyopia the demyelination is restricted to the optic nerve, while in Leber's hereditary optic atrophy it may be more widespread (Wilson
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