Abstract
The etiology of Alzheimer’s disease (AD) is complex, with both genetic and environmental factors influencing the expression of the phenotype. At present there are four wellestablished genes associated with AD. Mutations in the amyloid precursor protein (APP), presenilin 1 ( PSEN1), and presenilin 2 (PSEN2) genes are known to cause autosomal dominant early-onset Alzheimer’s disease (EOAD) (Theuns
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