Abstract
The State of Spontaneous Intracranial Hypotension in 2020: A Mini-Review
Highlights
The history of intracranial hypotension dates back to the early 1900’s, when trephined patients with depressed scars were observed to show decreased pressures via lumbar puncture manometry[1]
The etiology for spontaneous intracranial hypotension (SIH) has been attributed to CSF leaks due to osteodiscogenic microspurs, rupture of spinal nerve root diverticula, or, more controversially, CSF-venous fistulae[5,6]
Much of the discussion in this mini-review applies to secondary intracranial hypotension, special emphasis will be made on the primary form due to its inherently increased challenge in recognition, diagnosis and treatment
Summary
The history of intracranial hypotension dates back to the early 1900’s, when trephined patients with depressed scars were observed to show decreased pressures via lumbar puncture manometry[1]. Recognition of intracranial hypotension and its associations with CSF leak secondary to trauma, overshunting, lumbar puncture and surgery gradually increased[3,4]. In the case of intracranial hypotension, CSF loss results in compensation by increasing the volume of the venous compartment, given its increased compliance, leading to venous engorgement[11].
Sign-in/Register to view highlights & summary Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
