Abstract
This hypothesis attempts to explain (1) the spread of pulmonary tuberculosis as a vascular pressure phenomena. (2) Why pulmonary tuberculosis first appears in the area of the pulmonary broncheal anastomosis. (3) Why extensive infection in this area results in lowering the arterial pressure. (4) why when mitral stenosis is present it results in the slowing down of the tubercular process. (5) Why during preganacy, pressure on the abdominal aorta results in the improvement of the patient and why after delivery, this improvement ceases. (6) A possible explanation of why the right lung is attacked first in apical tuberculosis. (7) The part these vascular forces play when congenital stenosis of the pulmonary orifice is present.
Sign-in/Register to access full text options 
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
