Abstract
Under standardized conditions, circulating catecholamines (CA) can give a valid indication of the sympathetic status in human. This approach has permitted to uncover that 50% of labile hypertensive (HT) patients and 30% of stable HT patients had elevated circulating CA in supine position. These increased levels were mainly due to increased NE in stable HT and to increased E in labile HT. On that basis, the HT population could be subdivided in hyperadrenergic (elevated CA levels) and normoadrenergic (normal CA levels) subgroups. The hyperadrenergic subgroups were also characterized by a greater rise in CA and NE upon standing, by a faster heart rate, by an increased myocardial contractility and by a greater hypotensive response to treatment with beta-blockers.
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