The severity of pulmonary lesions in acquired heart defects

Abstract

Acquired heart defects are a group of diseases (represented by stenosis, valve insufficiency, combined and concomitant defects), accompanied by a violation of the structure and functions of the valvular heart apparatus and leading to changes in the intracardiac circulation. Against the background of such violations, stagnation occurs not only in the small, but also in the great blood circulation circle, which is manifested by a chronic dry cough, often with an admixture of blood. Compensated heart defects can be hidden, while decompensated ones are manifested by shortness of breath, palpitations, fatigue, pain in the heart, and a tendency to fainting. In such conditions, there is an increase in pressure in the pulmonary artery, which ultimately can cause the development of pulmonary edema. Acquired heart defects are also a serious danger in terms of the development of heart failure, damage to internal organs (in particular, the liver and kidneys), the appearance of ascites, and if the course is unfavorable, the fatal outcome is possible.