Abstract
The rather meager scientific yields of genome-wide association studies (GWASs) in schizophrenia have been partly attributed to the heterogeneity of the disorder and to different diagnostic procedures. To address these concerns, investigators analyzed genomic and detailed, interview-based, clinical data from a GWAS and from two independent, well-characterized replication datasets. Disregarding clinical status, the researchers identified all independent sets of interacting single-nucleotide polymorphisms (SNP sets) across both case …
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