Abstract
Type II endoleak (EL-2) is the most common complication of endovascular aneurysm repair (EVAR), leading to continued sac growth with potential risk of rupture. The contribution of lumbar arteries (LAs) to continued sac growth from EL-2 is unclear. We tested the hypothesis that the lower LAs (L3 and L4) play a greater role than the upper LAs (L1 and L2) in continued sac growth due to their rich collateral network with the iliolumbar and deep circumflex iliac arteries.
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