Abstract
Background & aims: Duodenal mucosal HCO3– secretion is a key factor in epithelial protection against injury by the acid discharged from the stomach. The aim of this investigation was to elucidate the role of the chloride/base exchanger SLC26A6, which is highly expressed in duodenal brush border membrane, and the cystic fibrosis transmembrane regulator (CFTR) in basal and stimulated murine duodenal HCO3– secretion in vivo.
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