Abstract
Rat livers perfused with a medium devoid of red cells do not release remarkable amounts of potassium when O2 is substituted by N2 for 30 min, nor does 5×10−5 M 2,4 DNP much increase the K+ concentration in the perfusate. On the other hand, high concentrations of sodium fluoride (7×10−3 M) produce a pronounced K+ efflux and Na+ uptake. These findings suggest that a) the cation equilibirium is relatively insensitivite to transient O2 deficiency and b) the active alkali cation transport may be coupled to the glycolytic energy supply in the liver.
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