Abstract
Endometriosis impacts approximately 10% of women with symptoms of debilitating pain or infertility (1). Women with endometriosis (endo) have been shown to have altered peritoneal macrophage activation (2) and increased peritoneal recruitment (3). The primary aim of this study is to determine if the increase in macrophage recruitment in endo is due to abnormal chemotactic signals originating in the peritoneal fluid (PF) or inherent differences in monocyte response. To address this question, we used peripheral blood monocytes (PBM) to eliminate potential influences of the peritoneal environment.
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