Abstract
The Role of Antibodies to Steroid Hormones and Chemical Carcinogen Benzo[A]Pyrene in Preterm Prelabour Rupture of Membranes
Highlights
Infants born too early are of public health concern since complications associated with preterm birth (PTB) are the leading cause of children’s mortality and a major cause of morbidity and disability [1,2]
Preterm prelabour rupture of membranes (PROM) occurs in 40% of all spontaneous preterm deliveries. [3,4,5,6]
It was evident that the level of Ig A to E2 was considerably higher in women of group I than of group II (p=0,002)
Summary
Infants born too early are of public health concern since complications associated with preterm birth (PTB) are the leading cause of children’s mortality and a major cause of morbidity and disability [1,2]. The proportion of PTB has no tendency to decrease and remains at the level of 5-10% varying from region to region in different countries. In recent years it has increased as the result of new reproductive technologies [7]. The question about the pathogenesis of preterm PROM in pregnancy is considered from different angles. The violation of the autoimmune tolerance to own tissues can lead to a number of problems of reproduction including complications during pregnancy, PTB, recurrent miscarriage, infertility and malformations in the fetus [8]. As for medical scientific literature, a strong increase of antibodies to steroid hormones results in adverse pregnancy outcomes [9,10]. Preterm prelabour rupture of membranes (PROM) occurs in 40% of all spontaneous preterm deliveries
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