Abstract

Section I: Transport Properties and Permeability.- The Basis for Active Transport at the Blood-Brain Barrier.- Studies of Cerebral Capillary Endothelial Membrane.- Biochemical Characteristics of Cerebral Capillaries.- Characteristics of Some Monoamine Uptake Systems in Isolated Cerebral Capillaries.- Transport of Sodium and Potassium across the Blood-Brain Barrier.- Some Properties of Isolated Endothelial Cells in Culture.- Induction of ?-Glutamyl Transpeptidase in Isolated Cerebral Endothelial Cells.- Vascular Basement Membranes: Preparation and Properties of Material Isolated with the Use of Detergents.- The Indicator Extraction Technique. A Method for Studying the Blood-Retinal Barriers.- Central Adrenergic Regulation of Cerebral Microvascular Permeability and Blood Flow Anatomic and Physiologic Evidence.- Central Adrenergic Regulation of Cerebral Microvascular Permeability and Blood Flow: Pharmacologic Evidence.- Ultrastructural and Histochemical Studies of Cerebral Capillary Synapses.- Cyclic Nucleotide Systems in the Microcirculation of Mammalian Brain.- Section II: Perturbation of the Barrier.- Blood-Brain Barrier Transport During Anesthesia.- Factors which Determine Cerebrovascular Permeability in the Normal Brain and Following Osmotic Treatment.- Endothelial and Astrocytic Cell Membranes in Relation to the Composition of Cerebral Extracellular Fluid.- The Blood-Brain Barrier in Acute and Chronic Hypertension.- Changes in the Cerebral Vasculature after Hypertension and Trauma: A Combined Scanning and Transmission Electron Microscopic Analysis.- Abnormalities of the Cerebral Microcirculation after Traumatic Injury: The Relationship of Hypertension and Prostaglandins.- Freeze Injury and Repair of Cerebral Microvessels.- Alterations in Cerebral Blood Flow Immediately after Brief Periods of Stasis.- The Effect of Severe Hypoxia on Cerebral Glucose Flux.- Behaviour of the Blood-Brain Barrier Toward Biogenic Amines in Experimental Cerebral Ischemia.- Changes in Blood-Brain Transfer Parameters Induced by Hyperosmolar Intracarotid Infusion and by Metastatic Tumor Growth.- Normal and Altered Regional Cerebral Blood Flow and Solute Movement Across the Cerebral Capillary in Rat.

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