Abstract
Experiments based on recordings of the reflex response of the blood pressure in cats to mechanical stimulation of pressoreceptors in the wall of the small intestine show that different agents affecting tissue metabolism when introduced into the intestinal lumen and applied to its surface cause changes in the reactivity of the intestinal pressoreceptors. Solutions of epinephnine, glucose and glucose with insulin increase the pressor reflex from the intestinal pressoreceptors while solutions of monoiodoacetic acid and sodium fluoride (inhibition of glycolysis) as well as 2,4-dinitrophenol (inhibition of phosphorylation connected with respiration) decrease it to the point of total disappearance. Insulin acts in the same manner; lactic acid restores the reflex inhibited by monoiodoacetic acid.
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