Abstract
The aim of the study was to analyze the relationship between the angle of spinal curvature and surface electromyography (SEMG) amplitude of the erector spinae in young school-children. A total of 251 children aged 7–8 participated in the study. The analysis involved 103 (41%) children with scoliosis, 141 (56.17%) with scoliotic posture, and seven (3.0%) with normal posture. Body posture was evaluated using the Diers formetric III 4D optoelectronic method. Analysis of SEMG amplitude of the erector spinae was performed with the Noraxon TeleMyo DTS apparatus. A significant correlation was found between the angle of spinal curvature and the SEMG amplitude of the erector spinae. The most important and statistically significant predictor of the SEMG amplitude and scoliosis angle in the scoliosis group was the standing position, chest segment, right side. The largest generalized SEMG amplitude of the erector spinae occurred in both boys and girls with scoliosis. Impaired balance of muscle tension in the erector spinae can trigger a set of changes that create a clinical and anatomopathological image of spinal curvature.
Highlights
The causes of idiopathic scoliosis have not been explained to date
In the group of boys were significant group-related differences found in surface electromyography (SEMG) amplitude of the erector spinae
The greatest generalized SEMG amplitude of the erector spinae occurred in scoliosis
Summary
The causes of idiopathic scoliosis have not been explained to date. There is no universally accepted theory of scoliosis etiopathogenesis. There is a consensus that idiopathic scoliosis is multifactorial. Extensive literature in this field describes abnormalities at the level of systems, organs, tissues, cells, particles, and genes, without prejudging their primary or secondary nature [1]. Scoliosis is obviously a distortion of posture and body structure, it is the effect of its compensatory abilities, allowing the preservation of the position of the head and shoulder girdle over the pelvis [3]. It is justifiable to talk about etiological factors, and not about a specific theory, e.g., genetic or metabolic scoliosis [4]
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