Abstract
Dilated cardiomyopathy is one of the most common causes of the heart failure in childhood and can develop as a consequence of metabolic disorders such as fatty acid beta-oxidation disorders or amino acids metabolism disorders. In our report we present a case where in the course of deficiency of long-chain 3-hydroxyacyl-coenzyme A dehydrogenase the recovery from dilated cardiomyopathy resulted in hypertrophic cardiomyopathy.
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