Abstract
Myo-inositol (MI) and d-chiro-inositol (DCI), two second messengers of insulin in various insulin-dependent processes, are implicated in polycystic ovary syndrome (PCOS). MI and DCI exert distinct functions in insulin signaling. While MI is involved in the activation of glucose transporters and glucose utilization, DCI is mainly involved in the glycogen synthesis. At ovarian level, MI plays a pivotal role in the FSH signaling while DCI promotes insulin-dependent androgen synthesis. Although MI and DCI fulfill different roles, often their biological activities are confused.
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