Abstract

Two models of gynodioecy controlled by cytoplasmic factors and nuclear genes are studied. In one model, there is a male-sterility determining cytoplasm which produces its effect only in the absence of a restorer allele at a nuclear locus. In the other model, either the cytoplasmic factor or the nuclear gene can produce male-sterility independently of the other. Neither model permits stable coexistence of the two types of factor in a population.

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