Abstract
Background: Diabetic peripheral neuropathy (DPN) involves both sensory and motor nerves; from pathological point of view it can cause demyelinating neuropathy (evident by increase in latency and decrease in conduction-velocity [CV]) as well as axonal degeneration neuropathy (evident by decrease in amplitude) of compound action potential. However there is controversy on which is the most affected nerve and nerve conduction study (NCS) parameter in DPN.
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