Abstract

Psoriasis is a chronic, recurrent and inflammatory skin disease. It is characterized by erythematous scaly patches on knees, elbows, sacral area and scalp. The etiopathogenesis of psoriasis is still unknown. Recent reports have suggested a role for the alternative complement pathway 1. Psoriasis may result from the interaction of yeasts and other microorganisms with an abnormally responsive alternative complement pathway 1,2. C. albicans is a potent activator of alternative complement pathway. This suggested that the yeast flora of gastrointestinal tract may play an important role in the etiopathogenesis of psoriasis.

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