Abstract
Renal dysfunction is a common complication following orthotopic heart transplantation (OHTx) that may be worsened by the early initiation of calcineurin inhibitors (CNI), especially in patients with pre-existing renal impairment. As a result, delayed initiation of CNI or CNI-free immunosuppression regimes are sometimes unavoidable, and temporizing strategies involving anti-thymocyte globulin (ATG) or basiliximab are used as an adjunct to immunosuppressive protocols. However, the most effective strategy for preventing acute cellular rejection (ACR) in this context is uncertain.
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