Abstract
1. 1. The neural control of the release of hyperlipaemic hormone has been studied in isolated corpora cardiaca of Locusta migratoria using electrical stimulation of the nervi corporis cardiaci I and II, and assaying the bathing medium for hyperlipaemic activity. 2. 2. Stimulation of nervus corpus cardiacum II (NCC II) results in the release of hormone which is dependent upon the arrival of compound action potentials within the glandular lobe of the corpus cardiacum. 3. 3. Sodium deficient Ringer and Ringer containing 10 −6 M tetrodotoxin abolish the compound action potential and with it the release of hormone. 4. 4. The mechanism of release is dependent upon the presence of extracellular calcium-ions. 5. 5. Stimulation of nervus corpus cardiacum I (NCC I) alone does not result in the release of hyperlipaemic hormone, but the presence of action potentials in NCC I potentiates the hyperlipaemic effect obtained by NCC II stimulation.
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