Abstract
Female sexual behavior was observed in ovariectomized female rats treated with varying doses of estrogen and progesterone, following electrolytic midbrain lesions. Severe deficits in receptivity were produced by lesions aimed at the dorsal and ventral norepinephrine pathways (DV lesions) and by lesions intended to destroy meso-limbic (A-10) dopamine-producing cells. Significant but less profound impairments were seen following lesions aimed at the serotonin-producing raphe nucleus or the nigro-striatal dopamine cells. Systemically administered d-amphetamine was found to increase receptivity in severely impaired A-10 and DV animals. The observed deficits may result from damage to midbrain structures involved in the sensory-motor or hormonal mediation of the lordosis reflex, or from disruption of a neurochemical system involved in control of lordosis.
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