Abstract

This article highlights the main factors of the pathogenesis of ischemia/reperfusion syndrome of renal allograft. Cellular, humoral, and nonspecific mechanisms of renal damage development are described. The possibilities of effective influence on it are limited by objective difficulties, which are mainly associated with the presence of a variety of alternative ways, which ultimately lead to severe graft damage, the rapid development of chronic transplant nephropathy and increase the risk of graft loss. Further research is needed to develop ways to target the main links of pathogenesis.

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