Abstract
Aim: The Vascular Smooth Muscle Cells (VSMCs) are exclusive cells. They have diverse function and generally accounted for vasomotor regulation and extracellular matrix synthesis. In response to patient hemodynamic status, neurohumoral stimuli and pathological conditions, the VSMCs transcriptome undergo substantial modulation leading to disbalance between contractile to synthetic phenotypes. Understanding of the regulatory role of miRNAs and targeted genes in the process of phenotype shift may help to control phenotypic modulation in pathological conditions.
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