Abstract
Cavernous angiomas of the central nervous system are rare, neurovascular lesions usually detected in patients aged 20-50 and represent up to 8-15% of all intracranial malformations. The association between cavernomas and arteriovenous malformations is met in 10-30% of cases. Treatment strategies for intracranial cavernomas include conservative management, open microsurgery and finally stereotactic radiosurgery (the use of which is still highly debatable as there are no available methods to determine its short term effects). The authors present their surgical experience in what regards a cohort of 149 consecutive patients, diagnosed with intracranial and supratentorial cavernomas, admitted, investigated and treated between January 2000 and January 2015. The clinical particularities of the patients together with the surgical approaches are debated. Last but not least the outcome and quality of life of the patients is discussed.
Highlights
IntroductionEven though Bremer and Carson [2] were the first who managed to perform a surgical resection of an intracranial cavernoma, Walter Dandy (1886-1946) was the first to successfully publish a series of operated patients while taking note at the same time of the clinical features of these lesions
Cavernous angiomas of the central nervous system are rare, neurovascular lesions usually detected in patients aged 20-50
Even though Bremer and Carson [2] were the first who managed to perform a surgical resection of an intracranial cavernoma, Walter Dandy (1886-1946) was the first to successfully publish a series of operated patients while taking note at the same time of the clinical features of these lesions
Summary
Even though Bremer and Carson [2] were the first who managed to perform a surgical resection of an intracranial cavernoma, Walter Dandy (1886-1946) was the first to successfully publish a series of operated patients while taking note at the same time of the clinical features of these lesions. It was Dandy who noticed for the first time that intracranial cavernomas are predisposed to bleeding, epileptic seizures and focal neurological deficits. The lesions become symptomatic when the size is larger than 1 cm
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