Abstract
Genomic imprinting can be regarded as one of many variants of epigenetic modes of gene regulation in eukaryotic cells. There is no a priori reason, therefore, to invoke fundamentally novel mechanisms to explain the imprinting phenomenon in mammals. For example, the different factors involved in imprinting, such as the stable propagation of different chromatin states that repress or permit gene transcription, are well-known entities in a wide range of eukaryotic cells (see Pirotta Chap. 10, and Geramisova and Corces Chap. 11, this vol.). From this point of view, we should perhaps consider the mechanism(s) of imprinting as being the unusual result of combinatory events that occur regularly on an evolutionary scale.
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