Abstract
The apoL1 C-terminal variants G1 and G2 are linked to CKD.[1][1] Expression of these variants induces effacement of podocyte foot processes, leading to the loss of these cells from glomeruli and impairment of the blood filtration activity of the kidney. A characteristic of G1/G2-linked disease is
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Published version (
Free)
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callMore From: Journal of the American Society of Nephrology : JASN
Paper Title
Journal
Date
