Abstract
Ectopic pregnancy (EP) can be divided into abortion of ectopic pregnancy (AEP) and rupture of ectopic pregnancy (REP) depending on the outcome of the pregnancy. REP not only leads to an increase in emergency surgery, loss of the diseased fallopian tube, and impairment of future natural fertility, but also poses a serious threat to maternal life. However, the mechanism of REP is still unclear. We have shown that extravillous trophoblast (EVT) over-invasion may be associated with the development of REP, and we have also verified that EVT is more invasive in REP than in AEP, further suggesting that EVT over-invasion plays an important role in REP. However, the regulatory mechanism of EVT biological behavior at the EP maternal-fetal interface is not clear so far.
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