Abstract
1. Hypoglycemia leads to a general decrease in cortical activity, measurable by a delay in and an inaccuracy of mental functions. This condition is accompanied by typical changes in the electroencephalogram and in the electrocorticogram which indicate a diminution in the oxidative state of the cortex (Hoagland). 2. Insulin hypoglycemia produces a state of increased excitability of sympathetic centers. This is shown by an increased blood pressure response to anoxia and to CO2 at low blood sugar levels. It is further indicated by the increased blood pressure rise on raised intracranial pressure during the hypoglycemic state. Since the increased response to CO2 and to increased intracranial pressure persists after elimination of the chemoreceptors of the carotid sinus area and of the arch of the aorta, it follows that low blood sugar increases sympathetic responses not only in response to reflex stimuli but also to stimuli acting directly on the centers. Another indication of increased excitability of the sy...
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