Abstract
Three indole acids accelerated glucose uptake in the rat diaphragm. The effect of indole butyric acid was greater than that of indole propionic, which in turn was greater than that of indole acetic. The in vitro hypoglycemic potency of the indole acids paralleled their in vivo potency. Increased hypoglycemic potency may be related to the number of carbon atoms in the indole acid side chain. These and earlier observations in experimental and human neoplasia associated with hypoglycemia suggest that the neoplasm is a rich source of tryptophan material which, in the bloodstream, produces hypoglycemia by increasing the glucose uptake in peripheral muscles, and possibly in the tumor itself.
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