Abstract
PRELIMINARY TO ITS USE IN THE STUDY of the thyrotoxic state in the human, the galactose tolerance test, as originally advocated by Althausen (1) was employed J L as a unit of measurement in dogs. Following Althausen's report, Maclagan and Rundle (2) as well as King et al (3, 4) employed the same type of test but they placed a different interpretation on their findings. Briefly, the American authors ad-vanced the explanation that the thyroid hormone increases the rate of absorption of galactose from the intestinal tract (5). In support of this concept, they quoted the work of Soskin et al. (6). The English authors (2, 3, 4) contended that the increase of the galactose in the circulating blood in the thyrotoxic state was due to liver damage; that the blood was therefore not cleared sufficiently rapidly of the galactose which was absorbed from the intestinal tract.(14).
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