Abstract
The pelvic endometriosis is considered a relatively common, benign, estrogen-dependent disease associated with complaints of pelvic pain and infertility, affecting 6% to 10% of women of reproductive age. The etiopathogenesis of the disease is based on some theories and hypotheses. Classical theories include the hypothesis of migratory pathogenesis by retrograde menstrual flow, the theory of celomic metaplasia, the theory of embryonic rest or even iatrogenic deposition during abdominal surgical procedures
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