The Impact of Resistance Exercise on Neurotransmission Failure within Trained Participants

Michael J. Maclellan ,Christopher M. Hearon ,Tyler M. Farney ,Arnold G. Nelson

doi:10.29199/2637-7039/boms-101017

Abstract

The Impact of Resistance Exercise on Neurotransmission Failure within Trained Participants

Highlights

Human performance is largely dependent upon both force and power generation, fatigue begins to set in during sustained muscle contractions [1]
Similar to the results presented by Aagaard et al, [26] it was hypothesized that M-wave amplitude would increase from pre- to post-exercise, with a greater increase being apparent following multiple repetitions
The main effect for exercise was not statistically significant (p = 0.7590, η2 = 0.014) where the M-wave amplitude for CR1 (5.50 ± 6.16 mV) and CR5 (4.88 ± 5.59 mV) were similar when pooled across pre-/post-exercise

Summary

Introduction

Human performance is largely dependent upon both force and power generation, fatigue begins to set in during sustained muscle contractions [1]. Fatigue is a multi-faceted process, the loss in force/power following submaximal contractions with sufficient rest between each muscle action has been more closely attributed to the alterations in the peripheral nervous system [5]. As for central nervous system fatigue, factors may include a progressive reduction in the voluntary activation of a muscle starting from the motor cortex [12] to the desensitization of moto neurons at the spinal level [13]. Central fatigue may result from an increased inhibition from group III and IV afferents [14]. The purpose of this investigation was to determine neuromuscular failure via pre-to-post exercise amplitude changes of the M-wave following a Calf-Raise Exercise (CR)

Methods

Results

Discussion

Conclusion