Abstract
Progressive supranuclear palsy (PSP) is a 4-repeat (4R) tauopathy, traditionally associated with Richardson's syndrome, which manifests with supranuclear gaze palsy and frequent falls due to postural instability [1]. Clinicopathological studies however have highlighted the great phenotypical variability underlying PSP, which may manifest with atypical syndromes, such as PSP with predominant Parkinsonism and primary gait freezing among others [1–3]. Moreover, some PSP patients may initially exhibit only subtle ophthalmokinetic abnormalities or normal postural reflexes.
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