Abstract
In 1957, while studying the pathogenesis of Mycoplasma synoviae, Dr. Norman Olson et al. (22), at West Virginia University, reported the isolation of an agent producing synovitis from lesions in broilers that exhibited a lack of sensitivity to chlortetracycline and furazolidone. Olson reported in 1959 (23) that this particular agent was not susceptible to streptomycin either, and Kerr and Olson (13) also observed that the newly found synovitis agent was pathogenic only in young chicks, whereas such age resistance was uncommon for M. synoviae infections. Subsequently, Olson and his coworkers (25) determined that the synovitis agent was a virus, which they called viral arthritis agent. It was first misdiagnosed as a poxvirus (25) because of its double-stranded nucleic acid. How-
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