Abstract
The mechanism of thrombus formation is a coordinated process that is under the control of neurohumoral regulation and is influenced by a variety of external and internal factors. Any disruption in the regulatory system, activation of some factors and inhibition of others can lead to disruption in the coagulation system, affecting both the plasma and platelet links of hemostasis. The activation of platelets against the background of the existing atherosclerosis of the vessels leads to the formation of blood clots, which clog the lumen of the coronary vessels and can lead to acute myocardial infarction. Today it is well known that increased thrombus formation is not only the cause of the development of acute conditions, but also leads to the progression of the disease. The leading role in the initiation of the thrombus formation process is played by platelets, therefore, the use of antiplatelet drugs is an obligatory link in both the treatment and prevention of thrombosis. In this case, the most important is the need to restore blood flow in the coronary vessels, which provide nutrition to the myocardium. In this regard, correctly selected antithrombotic agents can take a worthy place in the treatment and prevention of cardiovascular pathology, which is based on ischemic thrombosis.
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