Abstract
When rT1 infects Sh(P1), the release of modified uT1 by the rare yielder cells is delayed in a portion of these cells for at least 1 hour. In the nonyielding cells the Hr gene persists in an inactive, nonmultiplying state for some time, but after several hours of bacterial multiplication its presence cannot be demonstrated. Even in Sh(P1) cells jointly infected by rT1 and uT1, the genes from rT1 neither function nor replicate, unless they are rescued by recombination with uT1. Two strains of rT1, which have become restricted independently, fail to cooperate in the infection of Sh(P1).
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