Abstract
Hyperammonemic encephalopathy secondary to heart failure is rare and there had been little reports about effective treatment. Organ hypoperfusion or congestion by heart failure may lead to various organ dysfunctions, and liver and intestinal circulatory impairment might cause ammonia metabolic failure. Here, we report on the case of a patient with hyperammonemic encephalopathy that was secondary to heart failure, which was effectively treated by lactulose.
Highlights
Heart failure is associated with various organ dysfunctions such as hepatic or renal dysfunctions due to the impairment of organ perfusion [1,2]
Hyperammonemic encephalopathy developed in a patient with a medically intractable
Hyperammonemic encephalopathy developed in a patient with a medically intractable heart failure, who had no other apparent liver disease other in than congestion
Summary
Heart failure is associated with various organ dysfunctions such as hepatic or renal dysfunctions due to the impairment of organ perfusion [1,2]. Hepatic dysfunction in congestive heart failure is occasionally encountered, revealing some aspects of the condition [3]. There are few cases where complications due to hyperammonemic encephalopathy occurs. Hypoperfusion or congestion due to heart failure may lead to increased abdominal and liver damage, which might lead to the failure of ammonia metabolic balance [6]. There are scanty reports on cases of hyperammonemic encephalopathy associated with heart failure. We report on a case of heart failure with hyperammonemic encephalopathy, which was treated successfully by the addition of lactuloses
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